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Following a median follow-up of 2.1 years, the risk of suffering a major cardiovascular adverse event (death, nonfatal AMI, nonfatal stroke or hospitalization for unstable angina) was similar in both the lixisenatide and the placebo arms (HR 1.02
Exendin-4 also promotes the polarization of bone marrow-derived macrophages in mice towards the M2 subtype by modulating the STAT3 pathway and cAMP protein kinase A (PKA) signaling [91, 92]
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